A Case-Based Guide to Clinical Endocrinology by Steven Jon Russell, Karen Klahr Miller (auth.), Terry F. PDF

By Steven Jon Russell, Karen Klahr Miller (auth.), Terry F. Davies (eds.)

ISBN-10: 1588298159

ISBN-13: 9781588298157

ISBN-10: 1603271031

ISBN-13: 9781603271035

Written instances supply a considerate assessment of contemporary administration and contemplation of the literature, even more so than even a case presentation with out the sufferer. accordingly, lately the written or ready case has develop into the appropriate discussion board for demonstrating how one can deal with a clinical challenge to the best good thing about the sufferer. In A Case-Based consultant to medical Endocrinology, amazing clinicians supply stimulating guideline and insights right into a good selection of endocrine matters, educating readers glossy administration of the stipulations defined and supplying designated access into the literature. Importantly, a number of the very important components within the present curriculum for scientific endocrine education as advised through the organization of application administrators in medical Endocrinology, Diabetes and Metabolism are coated during this textual content. to augment studying and retention of information, every one writer has additionally supplied a number of selection questions following the case reports. scholars, citizens, fellows, and practitioners will locate those examples to be an effective solution to evaluate their knowing and bring up their talents and skills in sufferer research and administration.

In all, A Case-Based consultant to medical Endocrinology bargains a entire quantity of concise case-studies that might turn out to be of important value to all clinicians working towards during this box.

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True or false? 2. They invariably cause amenorrhea and galactorrhea in women. True or false? 3. The best treatment of microprolactinomas is A. Medical with dopamine agonists B. Surgical C. Both 4. Withdrawal from dopamine-agonist is invariably followed by recurrent hyperprolactinemia. True or false? Chapter 3 Cushing’s Disease Kawaljeet Kaur and James W. Findling Objectives To identify the patient population that should be screened for hypercortisolism, and to understand the approach to the diagnosis, differential diagnosis, and management of Cushing’s syndrome.

Preoperative laboratory measurements revealed no evidence of elevated IGF-I or thyroid hormone. The prolactin was too low for a prolactinoma of this size, suggesting the elevation was likely due to pituitary stalk compression. The pathology showing necrosis and a population of cells staining for FSH, LH, and ␣-subunit confirmed the clinical diagnosis of apoplexy in a clinically nonfunctioning pituitary adenoma. Lessons Learned Pituitary apoplexy (apoplexy meaning “sudden attack”, or “to be struck down”) is a clinical syndrome in which the abrupt onset of typical signs and symptoms (see below) result from hemorrhage or infarction within the pituitary or a pituitary tumor.

Findling JW, Raff H. Screening and diagnosis of Cushing’s syndrome. Endocrinol Metab Clin North Am 2005;34(2):385–402, ix–x. Lindsay JR, Nieman LK. Differential diagnosis and imaging in Cushing’s syndrome. Endocrinol Metab Clin North Am 2005;34(2):403-421, x. Raff H, Findling JW. A physiologic approach to diagnosis of the Cushing syndrome. Ann Intern Med 2003;138(12):980–991. Utz AL, Swearingen B, Biller BM. Pituitary surgery and postoperative management in Cushing’s disease. Endocrinol Metab Clin North Am 2005;34(2):459-478, xi.

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A Case-Based Guide to Clinical Endocrinology by Steven Jon Russell, Karen Klahr Miller (auth.), Terry F. Davies (eds.)


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